Association of Human Papillomavirus Genotype 16 Viral Variant and Viral Load with Cervical High-grade Intraepithelial Lesions


Por: Alvarez-Paredes, L, Santibanez, M, Galiana, A, Diaz, J, Paras-Bravo, P, Andrada-Becerra, M, Garcia, M, Rodriguez-Ingelmo, J, Portilla-Sogorb, J and Paz-Zulueta, M

Publicada: 1 ago 2019
Resumen:
Human papillomavirus genotype 16 (HPV16) is by far the genotype most strongly associated with cervical cancer; viral variant and/or viral load of HPV16 could modulate this association. The objective was to determine the association between the viral variant and viral load of HPV16 and the presence of cervical high-grade lesions. This cross-sectional study included all women in whom HPV infection was found by cervical smear during routine gynecologic health checks. Women with single or multiple HPV16 infections (n = 176) were selected for viral variant and viral load analysis. Smear results were classified using the Bethesda system. HPV types were classified according to the International Agency for Research on Cancer. Odds ratios (OR) with their 95% confidence intervals (CI) were estimated by logistic regression, adjusted for age, immigrant status, and coinfection with other high-risk genotypes. No statistically significant associations were found regarding the detected viral variants. A viral load above the median (>1,367.79 copies/cell) was associated with a significant risk of high-grade epithelial lesion or carcinoma, after adjusting for age, immigrant status, coinfections, and viral variant: (adjusted OR 7.89; 95% CI: 2.75-22.68). This relationship showed a statistically significant dose-response pattern after categorizing by viral load tertiles: adjusted OR for a viral load greater than the third textile was 17.23 (95% CI: 4.20-70.65), with adjusted linear P-trend = 0.001. In patients infected with HPV16, viral load is associated with high-grade intraepithelial lesions or cervical carcinoma. This could be useful as prognostic biomarker of neoplastic progression and as screening for cervical cancer.

Filiaciones:
Alvarez-Paredes, L:
 Univ Hosp Burgos, Dept Microbiol & Parasitol, Burgos, Spain

Santibanez, M:
 Univ Cantabria, Nursing Res Grp IDIVAL, Dept Nursing, Cantabria, Spain

Galiana, A:
 Univ Gen Hosp Elche, Dept Microbiol, Alicante, Spain

:
 Univ Gen Hosp Alicante, Dept Microbiol, Alicante, Spain

Paras-Bravo, P:
 Univ Cantabria, Nursing Res Grp IDIVAL, Dept Nursing, Cantabria, Spain

Andrada-Becerra, M:
 Univ Gen Hosp Elche, Dept Pathol Anat, Alicante, Spain

Garcia, M:
 Univ Gen Hosp Elche, Dept Microbiol, Alicante, Spain

Rodriguez-Ingelmo, J:
 Univ Gen Hosp Elche, Dept Gynecol, Alicante, Spain

Portilla-Sogorb, J:
 Univ Hosp Alicante, Dept Infect Dis, Alicante, Spain

Paz-Zulueta, M:
 Univ Cantabria, Dept Nursing, Cantabria, Spain

 IDIVAL, GRIDES, GI Derecho Sanitario & Bioet, Cantabria, Spain
ISSN: 19406207





CANCER PREVENTION RESEARCH
Editorial
American Association for Cancer Research, 615 CHESTNUT ST, 17TH FLOOR, PHILADELPHIA, PA 19106-4404 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 12 Número: 8
Páginas: 547-555
WOS Id: 000478016000006
ID de PubMed: 31208965
imagen Open Access

MÉTRICAS